fhs74 int Search Results


fhs74int human small intestine epithelial cells  (ATCC)
95
ATCC fhs74int human small intestine epithelial cells
Signaling events induced by FGF-23–Klotho. Immunoblotting showing that FGF-23 or Klotho alone have no effect on kinase activity in PTEC or <t>FHs74Int</t> cells. Combined effects of FGF-23 and Klotho show increased phosphorylation of ERK1/2, p38, JNK, AKT, IκB, and GSK-3β. α-Tubulin was used as a loading control.
Fhs74int Human Small Intestine Epithelial Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/fhs74int human small intestine epithelial cells/product/ATCC
Average 95 stars, based on 1 article reviews
fhs74int human small intestine epithelial cells - by Bioz Stars, 2026-03
95/100 stars
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fhs 74 int cells ccl-241  (Cedarlane)
90
Cedarlane fhs 74 int cells ccl-241
Signaling events induced by FGF-23–Klotho. Immunoblotting showing that FGF-23 or Klotho alone have no effect on kinase activity in PTEC or <t>FHs74Int</t> cells. Combined effects of FGF-23 and Klotho show increased phosphorylation of ERK1/2, p38, JNK, AKT, IκB, and GSK-3β. α-Tubulin was used as a loading control.
Fhs 74 Int Cells Ccl 241, supplied by Cedarlane, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/fhs 74 int cells ccl-241/product/Cedarlane
Average 90 stars, based on 1 article reviews
fhs 74 int cells ccl-241 - by Bioz Stars, 2026-03
90/100 stars
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human small intestinal epithelial cell line fhs74 int  (Procell Inc)
90
Procell Inc human small intestinal epithelial cell line fhs74 int
Signaling events induced by FGF-23–Klotho. Immunoblotting showing that FGF-23 or Klotho alone have no effect on kinase activity in PTEC or <t>FHs74Int</t> cells. Combined effects of FGF-23 and Klotho show increased phosphorylation of ERK1/2, p38, JNK, AKT, IκB, and GSK-3β. α-Tubulin was used as a loading control.
Human Small Intestinal Epithelial Cell Line Fhs74 Int, supplied by Procell Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/human small intestinal epithelial cell line fhs74 int/product/Procell Inc
Average 90 stars, based on 1 article reviews
human small intestinal epithelial cell line fhs74 int - by Bioz Stars, 2026-03
90/100 stars
  Buy from Supplier
fhs 74 int  (Leusch GmbH)
90
Leusch GmbH fhs 74 int
Signaling events induced by FGF-23–Klotho. Immunoblotting showing that FGF-23 or Klotho alone have no effect on kinase activity in PTEC or <t>FHs74Int</t> cells. Combined effects of FGF-23 and Klotho show increased phosphorylation of ERK1/2, p38, JNK, AKT, IκB, and GSK-3β. α-Tubulin was used as a loading control.
Fhs 74 Int, supplied by Leusch GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/fhs 74 int/product/Leusch GmbH
Average 90 stars, based on 1 article reviews
fhs 74 int - by Bioz Stars, 2026-03
90/100 stars
  Buy from Supplier

Image Search Results


Signaling events induced by FGF-23–Klotho. Immunoblotting showing that FGF-23 or Klotho alone have no effect on kinase activity in PTEC or FHs74Int cells. Combined effects of FGF-23 and Klotho show increased phosphorylation of ERK1/2, p38, JNK, AKT, IκB, and GSK-3β. α-Tubulin was used as a loading control.

Journal: The Journal of Cell Biology

Article Title: FGF-23–Klotho signaling stimulates proliferation and prevents vitamin D–induced apoptosis

doi: 10.1083/jcb.200803024

Figure Lengend Snippet: Signaling events induced by FGF-23–Klotho. Immunoblotting showing that FGF-23 or Klotho alone have no effect on kinase activity in PTEC or FHs74Int cells. Combined effects of FGF-23 and Klotho show increased phosphorylation of ERK1/2, p38, JNK, AKT, IκB, and GSK-3β. α-Tubulin was used as a loading control.

Article Snippet: Human renal PTEC were obtained from Clontech Laboratories, Inc. FHs74Int human small intestine epithelial cells were acquired from the American Type Culture Collection.

Techniques: Western Blot, Activity Assay, Phospho-proteomics, Control

FGF-23–Klotho prevents vitamin D–induced apoptosis. (A) ELISA analysis of 1α-hydroxylase expression showing no significant changes in PTEC cells exposed to FGF-23 or Klotho alone but greatly decreased levels when exposed to both FGF-23 and Klotho. Small molecule inhibitors against Ras and PI3K were sufficient to provide marginal rescue of this decrease in expression. No significant changes were found for treatment of FHs74Int cells. Graphs represent mean ± SD ( n = 3). *, P < 0.05. (B) Flow cytometry analysis for TUNEL staining of cells exposed to exogenous vitamin D showing that it caused extremely high levels of apoptosis. Addition of FGF-23 and Klotho was sufficient to rescue most of the vitamin D–induced apoptosis, whereas FGF-23 or Klotho alone did not. PI3K inhibitor prevented this rescue, whereas Ras inhibitor had no effect. (C) ELISA for active caspase-3 levels, showing the same patterns as observed with the TUNEL analysis. Graphs represent mean ± SD ( n = 3). *, P < 0.001.

Journal: The Journal of Cell Biology

Article Title: FGF-23–Klotho signaling stimulates proliferation and prevents vitamin D–induced apoptosis

doi: 10.1083/jcb.200803024

Figure Lengend Snippet: FGF-23–Klotho prevents vitamin D–induced apoptosis. (A) ELISA analysis of 1α-hydroxylase expression showing no significant changes in PTEC cells exposed to FGF-23 or Klotho alone but greatly decreased levels when exposed to both FGF-23 and Klotho. Small molecule inhibitors against Ras and PI3K were sufficient to provide marginal rescue of this decrease in expression. No significant changes were found for treatment of FHs74Int cells. Graphs represent mean ± SD ( n = 3). *, P < 0.05. (B) Flow cytometry analysis for TUNEL staining of cells exposed to exogenous vitamin D showing that it caused extremely high levels of apoptosis. Addition of FGF-23 and Klotho was sufficient to rescue most of the vitamin D–induced apoptosis, whereas FGF-23 or Klotho alone did not. PI3K inhibitor prevented this rescue, whereas Ras inhibitor had no effect. (C) ELISA for active caspase-3 levels, showing the same patterns as observed with the TUNEL analysis. Graphs represent mean ± SD ( n = 3). *, P < 0.001.

Article Snippet: Human renal PTEC were obtained from Clontech Laboratories, Inc. FHs74Int human small intestine epithelial cells were acquired from the American Type Culture Collection.

Techniques: Enzyme-linked Immunosorbent Assay, Expressing, Flow Cytometry, TUNEL Assay, Staining